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Researchers believe this effect may involve: Increased epithelial cell migration Enhanced tissue regeneration Protection of intestinal lining integrity These mechanisms may explain why the peptide has been studied as a possible therapeutic agent for gastrointestinal injury models
these changes do not make it a growth-hormone secretagogue, instead they sharply reduce its binding to IGF-binding proteins (IGFBPs), which is what extends its circulating half-life and potency relative to native IGF-1
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Long-term clinical and MRI results of matrix-assisted autologous chondrocyte implantation for articular cartilage defects of the knee
Caveolin-1 promotes gastric cancer progression by up-regulating epithelial to mesenchymal transition by crosstalk of signalling mechanisms under hypoxic condition
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