Other loci, such as glucokinase regulator (GCKR) and membrane bound O-acyltransferase domain-containing 7 (MBOAT7), influence glucose metabolism, phospholipid remodeling, and insulin sensitivity, highlighting the tight link between metabolic dysfunction and liver injury
TSH (thyroid) and HbA1c (blood sugar) on file
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We study the effect of GSH and sexual dimorphism on acetaminophen toxicity by using a previously constructed model of acetaminophen metabolism [10]
Tripeptide in human plasma that prolongs survival of normal liver cells and stimulates growth in neoplastic liver. Nature New Biology , 243(124), 8587