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The interest in tendon recovery is backed by preclinical work such as this study examining how BPC-157 influences tendon fibroblast healing and growth factor expression in injured connective tissue

Additionally, changes in the microenvironment, such as redox state, altered pH, and the presence of reactive oxygen species (ROS) at the pathophysiological sites, can act as sensitive stimuli to trigger drug release in those areas

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PGC-1 also co-activates ERRs to enhance mitochondrial biogenesis and glucose utilization (Singh et al., 2018)