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Revisiting the role of phospholipases C in virulence and the lifecycle of Mycobacterium tuberculosis
Free radicals can damage renal tubular cells and disrupt the function of sodium transporters, leading to sodium retention and increased blood volume, which further elevates blood pressure [82]
73, S477S485
Still, confirming the clinical relevance of newly developed antioxidants will require a paradigm shift in the testing approaches
Activating upstream targets of the GSH system The key upstream target for GSH system modulation is generally considered to be Nrf2, a redox-sensitive regulator
Macrophage extracellular traps aggravate iron overload-related liver ischaemia/reperfusion injury
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