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LPS induction of a second group of genes (Prdx1, Srxn1, Hmox1, GSH synthase, cysteine transporters), mapping to nrf2 and the oxidative stress response, was increased by GSH depletion
In this way, GLP-1 RAs exert cardiovascular and renal protective effects, improving nonalcoholic fatty liver disease through mechanisms including weight loss, blood glucose control, blood pressure reduction, lipid improvement, insulin resistance improvement, and inflammation and oxidative stress reduction [8]
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M.LukA.et al
Vitamin deficiencies, DNA changes, and blood vessel damage harm the placenta, raising the risk of high blood pressure, preeclampsia, and other complications for mother and baby
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