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Stronger effects observed in older adult populations may reflect unique pathophysiological mechanisms of late-life depression, including mitochondrial dysfunction and neurodegenerative processes, which ALC could plausibly target

Some compounds have strong mechanistic logic but thin clinical evidence

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- cAMP, -arrestin, Receptor Internalization MOA ( ) - (Semaglutide, Tirzepatide ) - , - Workflow Target: GLP-1 Receptor / GLP-1 Figure 1