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FIGURE 1 Inflammation Neuroinflammation is a key pathophysiological mechanism of secondary injury, as it can produce neurotoxic effects on neurons through oxidative stress, apoptosis, and excitotoxicity (Kumar and Loane, 2012), inducing a progressive cycle of chronic inflammation (Sulhan et al., 2020)

This indicates that GHK-Cu peptide effectively downregulated chemokine-related immune activation, supporting a more stable neuroinflammatory environment

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GLP-1-directed NMDA receptor antagonism for obesity treatment

The drug tested was Rybelsus, a pill approved for type 2 diabetes manufactured by Novo Nordisk

While TSH is a vital "boss" hormone, it doesn't always tell the whole story