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demonstrated that BPC-157 counteracted serotonin syndrome induced by the combination of a monoamine oxidase inhibitor (MAOI) and L-tryptophan in rats [6]

Strength and limitation This review skillfully combines the most recent findings on methylglyoxal (MG) as a crucial mediator that goes beyond glucose rise with the complex effects of hyperglycemia on diabetic neuropathy (DN), including its involvement in sensory loss and wound healing deficiencies

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