And it appears endogenous GLP-1 does just that, it helps locally and temporarily to help suppress T-cell inflammation, the problem is that the half-life of endogenous GLP-1 is just a few minutes, so any systemic effects are somewhat muted
So, meals are digested more slowly, which lowers the rise in blood sugar after a meal
Regulation of TGF- signal via vagal activity In a septic rat model, vagus nerve activity has been demonstrated to inhibit TGF- production under hormone treatment
This is true whether youre on a GLP-1 drug or not
The suppressing effect of GLP-1 on alcohol intake is linked to suppressed alcohol reward, an idea now tested in both rats and mice with two different tests of alcohol reward behavior, adding strength to the conclusion
Evening dosing avoids daytime impairment