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Our findings demonstrate that SLC25A39 plays a critical role in maintaining mitochondrial homeostasis and HCC progression, and its knockdown activates a caspase-dependent apoptotic pathway through the mitochondrial ROScytochrome c pathway, inhibiting the proliferation, migration, and invasion of HCC
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GHK-Cu treatment significantly mitigated this reduction in colon length
This may be an indicator of a high level of oxidative stress in non-structural cardiac arrhythmias
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