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Huang Y, et al

Microbiota-derived metabolites influence neuroinflammation, blood-brain barrier integrity, vagal signaling, and hypothalamic circuitry, suggesting that enhancing endogenous GLP-1 may complement pharmacologic GLP-1R agonism by engaging both peripheral and central metabolic pathways, warranting more evidence-based mechanistic insights in the future

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The active molecule responsible

In diabetic mice, rescuing PINK1-mediated mitophagy by caffeic acid phenethyl ester could protect TECs against ferroptosis (79, 200)

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