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Our findings reveal that OSR addresses fundamental pathogenic mechanisms of OA by restoring redox homeostasis through the GPX4/SLC7A11-GSH axis, thereby simultaneously attenuating inflammation and oxidative stress while promoting cartilage regeneration

Proper injection site rotation is essential regardless of frequency

Zhu X, Uetrecht J

Anal Biochem 143 :111116

Similarly, IL-1-induced BMSC-Exos reversed hemin-induced ferroptosis in neuronal cells, including high iron, ROS, malondialdehyde (MDA), 4-hydroxynonenal (4-HNE), and reduced GSH level and cell viability [143]