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PPP is often upregulated in tumors and their enzymes are frequently overexpressed in cancer [23,24]
This modification results in the accumulation of labile iron within the mitochondria, leading to iron-induced instability and cell death[185]
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In CRC, the reactivation or remodeling of mitochondrial metabolic pathways, particularly oxidative phosphorylation (OXPHOS) and fatty acid oxidation (FAO), is closely associated with key oncogenic processes such as the evasion of apoptosis, the maintenance of cancer stem cell properties, and the regulation of redox homeostasis [5,6,7,8]
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The exact mechanisms are still being studied, but early research suggests that BPC-157 promotes healing by interacting with growth factors and repairing damaged tissues at the cellular level
