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Pathophysiology: Results from a severe congenital (disproportionately higher representation in pregnancy vs non-pregnancy compared with acquired form) or acquired deficiency in ADAMTS13, the specific metalloproteinase that cleaves ultralarge (UL) multimers of von Willebrand factor (VWF), Thrombosis occurs in the placenta in untreated TTP pregnancies and results in fetal growth restriction, intrauterine fetal death and preeclampsia
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Additionally, abnormal RBCs membranes lose phospholipid asymmetry, and expose phosphatydyl serine (PS) both on their membranes and on RBC derived microparticles which are shed into the blood circulation 37
GLOW and GLP-1 medications act through different receptor systems
Mechanisms: It enhances physiologic GH pulsatility, particularly during sleep cycles, supporting recovery, protein synthesis, and tissue repair without overstimulation or endocrine disruption
