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Exposure to lead resulted in elevated intracellular Ca 2+ level, activation of -calpain, and ultimately externalization of PS in erythrocytes: the exposed workers showed 2.82% PS externalization compared to 0.1% for unexposed controls [302]

The improvement of this functional marker may indicate an important pathway towards new therapeutic strategies that target glutathione dysregulation in schizophrenia

In a study on Fe-deficient mice, researchers found these mice had lower pain thresholds and were more sensitive to pain, which parallels the pathophysiological traits of FM 1

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