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The authors demonstrate that GLP-1R agonist treatment induces expression of tyrosine kinase IGF1R/ErbB (EGFR) pathways, however, agonist treatment can still increase intestinal growth in the absence of intestinal epithelial IGF1 receptor as well as EGF receptor signaling (Koehler et al., 2015)

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Marso et al., 2016b

Cell Mol Life Sci 2005

CHCHD2 mutations in autosomal dominant late-onset Parkinsons disease: a genome-wide linkage and sequencing study

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