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Thus, our data suggest GLP-1RAs may enhance LDL-C clearance by increasing LDLR expression, potentially improving CVD outcomes, with LDLR regulation occurring through both SREBP-2-dependent and SREBP-2-independent pathways
the expression vector according to present invention comprises of promoters, selection marker, multiple cloning regions, origin of replication & operator /repressor system
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While there is mechanistic support for this (different receptor occupancy thresholds), it has not been definitively demonstrated in human studies
Thats not timidity
R., Borner, T
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