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This is the primary mechanism that researchers are investigating
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The molecule is permeable to cell membranes (confirmed by Caco-2 assay) and highly selective, without inhibitory effect on other SAM-dependent methyltransferases
nursingtimes.net/clinical-a
Reviews in Endocrine & Metabolic Disorders
[12] Gastric acid-suppressing agents (proton-pump inhibitors and H-receptor antagonists) diminish dietary B12 liberation from protein complexes, a pharmacodynamic interaction that can exacerbate underlying deficiency
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