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We also saw more reports of hypersensitivity-type reactions, such as urticaria, than we anticipated, which raises interesting questions about possible immune-related mechanisms. Researchers also calculated the proportional reporting ratio (PRR) comparing adverse event rates between GLP-1 receptor agonist and DPP-IV inhibitors

Although the mechanisms whereby TNBS cause an inflammatory response are not completely defined, this response is thought to be initiated by macrophage-mediated recognition and degradation of the TNBS-modified cells and proteins within the colonic interstitium (Grisham et al, 1991