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While the body has systems in place to repair damaged cells, persistent oxidative stress overwhelms those mechanisms, setting off a chain reaction of cellular dysfunction

Each pathway contributes something distinct: GLP-1 reduces appetite and slows gastric emptying, GIP enhances insulin secretion and energy partitioning, and glucagon receptor activation increases energy expenditure and fat oxidation

The main risk is that a condensed eating window makes it harder to hit protein targets

Whatever it takes to keep fluids moving through your system

More fat means more conversion, leaving less testosterone available

Until such data are available, current evidence suggests a neutral to potentially favorable association, but clinicians should avoid overstating conclusions