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In particular, diabetes-related hyperglycemia acts through oxidative stress, fibrosis and severe inflammation in the reproductive tract, as demonstrated by the increase in serum transforming growth factor beta (TGF-), malonylaldehyde (MDA), and pentraxin-3 (PTX-3), with reduction in ovarian reserve and antimullerian hormone (AMH)

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I n skeletal muscle, IGF1R activation sensitizes IRS1 to degradation, and a screen for the responsible E3 ligase identified Fbxo40 as mediating this rapid turnover of IRS1, since IRS1 loss can be rescued by knockdown of Fbxo40

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HK1 content increased upon Gclc -ablation in FoB and in Gclc fl/fl MZB (Supplementary Fig