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Another limitation of many of the cited studies was that although the majority of the authors gave justification for research (hypothesis specifically was not always outlined), they did not appear to critically evaluate their own methodologies
By antagonism of central 2 -adrenergic heteroreceptors and autoreceptors as well as postsynaptic inhibition of 5-HT2 and 5-HT3 receptors, MTZ increases noradrenergic and 5-HT1A-mediated serotonergic neurotransmission (Fig
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